Ataxia - A Medical Dictionary, Bibliography, and Annotated by Icon Health Publications PDF

By Icon Health Publications

ISBN-10: 0597843406

ISBN-13: 9780597843402

ISBN-10: 1417512601

ISBN-13: 9781417512607

This can be a 3-in-1 reference ebook. It provides a whole clinical dictionary protecting thousands of phrases and expressions with regards to ataxia. It additionally provides large lists of bibliographic citations. ultimately, it offers details to clients on find out how to replace their wisdom utilizing quite a few web assets. The e-book is designed for physicians, clinical scholars getting ready for Board examinations, scientific researchers, and sufferers who are looking to get to grips with learn devoted to ataxia. in case your time is effective, this publication is for you. First, you won't waste time looking the web whereas lacking loads of suitable info. moment, the booklet additionally saves you time indexing and defining entries. eventually, you won't waste time and cash printing countless numbers of websites.

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Recombinant congenic lines will be produced from this small interval to estimate the effect size of the locus, ALCP1. Multiple alcohol-related and other behaviors (possibly associated with high ethanol intake) will be assessed in this congenic strain to determine the pleiotropic effects of this locus. ; Associate Professor; Pediatrics; Indiana Univ-Purdue Univ at Indianapolis 620 Union Drive, Room 618 Indianapolis, in 462025167 Timing: Fiscal Year 2002; Project Start 25-APR-2001; Project End 31-MAR-2004 Summary: (Provided by applicant): In humans, hereditary cerebellar ataxias develop gradually resulting from the degeneration of cerebellar neurons and their afferent and efferent connections.

Specific Aims Aim 1 Generation of a "knock-in" mutant mouse harboring a missense mutation in Atm (S2602C) Aim 2 Investigation of predisposition to cancer development in mice heterozygous for Atm (S2602C) Aim 3 Investigation of the mechanism of dominant interference by the Atm (S2602C) missense mutation To address the issue of cancer predisposition in A-T heterozygotes, a mouse model expressing an Atm missense mutation (corresponding to S2592C already detected in this gene in a patient with breast cancer and shown to interfere with ATM kinase) will be generated and cancer development monitored in carriers of the defective gene.

Our long range goal is to help elucidate the mechanisms by which cerebellar damage alters the production of normal movement and provide information that will enhance rehabilitation treatments for ataxia. The proposed studies will test the general hypothesis that the cerebellum adjusts the relative movements between joints and limbs through trial-and-error practice, making movements automatic and efficient. We believe that deficits in automaticity and efficiency combine to make movements far more mentally and physically taxing, leading to a decline in general activity level and function.

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Ataxia - A Medical Dictionary, Bibliography, and Annotated Research Guide to Internet References by Icon Health Publications


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